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Differential Effects of Human L1CAM Mutations on Complementing Guidance and Synaptic Defects in Drosophila melanogaster
New Tools for Targeted Disruption of Cholinergic Synaptic Transmission in Drosophila melanogaster
Transsynaptic Coordination of Synaptic Growth, Function, and Stability by the L1-Type CAM Neuroglian
Expression of human amyloid precursor protein in the skeletal muscles of Drosophila results in age- and activity-dependent muscle weakenss