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Mutations of Cardiac Troponin I cause Restrictive Cardiomyopathy; is cTnI N-terminal deletion an effective rescuing approach ?
Dose-dependent arrhythmia and cardiac dysfunction in restrictive cardiomyopathy mice due to troponin mutations
Roles of planar cell polarity pathways in the development of neutral tube defects
Sodium valproate-induced congenital cardiac abnormalities in mice are associated with the inhibition of histone deacetylase
Spatiotemporal expression of histone acetyltransferases, p300 and CBP, in developing embryonic hearts
DNA methylation regulates mouse cardiac myofibril gene expression during heart development
Protective effects of indomethacin and dexamethasone in a goat model with intrauterine balloon aortic valvuloplasty
Evaluation of cardiac function in cTnI(R192H) transgenic mice and cTni knockout mice with High-Resolution Ultrasound Imaging and Doppler Echocardiography
Energy metabolism and slow skeletal troponin I phosphorylation in cardiac troponin I null mouse heart
N-terminal truncated cardiac TnI improves cardiac function in vivo and rescues restrictive cardiomyopathy
Green tea extract catechin improves internal cardiac muscle relaxation in RCM mice
Identification of a truncated form of methionine sulfoxide reductase a expressed in mouse embryonic stem cells
Myofibril-Inducing RNA (MIR) is essential for tropomyosin expression and myofibrillogenesis in axolotl hearts
Inhibition of Histone H3K9 Acetylation by Anacardic Acid Can Correct the Over-Expression of Gata4 in the Hearts of Fetal Mice Exposed to Alcohol during Pregnancy
cTnI N-Terminal deletion: an agent for rescuing restrictive cardiomyopathy, a disease caused by mutations of Cardiac Troponin I
Muscle Contractility and Cell Motility
Planar Cell Polarity Signaling Pathway in Congenital Heart Diseases